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| Ch. 1 | Models of Toxicity Screening Using Cultured Cells | 3 |
| Ch. 2 | Apoptosis: Cellular Signaling and Molecular Mechanisms | 25 |
| Ch. 3 | Role of Mitochondrial Injury during Oxidative Injury to Hepatocytes: Evidence of a Mitochondrial Permeability Transition by Laser Scanning Confocal Microscopy | 49 |
| Ch. 4 | Glycine Cytoprotection and Inhibition of Nonlysosomal Calcium-Dependent Proteolysis during Anoxic Injury of Rat Hepatocytes | 65 |
| Ch. 5 | Di-Calciphor-Dependent Protection against Cell Death due to Mitochondrial Failure | 79 |
| Ch. 6 | Role of the Mitochondrial ATPase Inhibitor, IF[subscript 1], in Cytoprotection during Ischemia in Slow and Fast Heart-Rate Hearts | 87 |
| Ch. 7 | The Discovery and Pharmacology of Tirilazad Mesylate | 105 |
| Ch. 8 | The Role of Neutrophils and Reactive Oxygen Metabolites in Reperfusion Injury | 117 |
| Ch. 9 | The Inflammatory Response after Hepatic Ischemia/Reperfusion | 127 |
| Ch. 10 | The pH Paradox in Reperfusion Injury to Heart Cells | 149 |
| Ch. 11 | Pathophysiology of Spinal Cord Injury in a Rat Model of Decompression Sickness | 163 |
| Ch. 12 | The Stress Response and Stress Proteins | 177 |
| Ch. 13 | Cross Talk between Heat Shock and Oxidative Stress Inducible Genes during Myocardial Adaptation to Ischemia | 193 |
| Ch. 14 | Alteration in Cell Cycle Control Factors and the Induction of Oxygen-Regulated Proteins by Hypoxic Stress | 213 |
| Ch. 15 | Stress Proteins in Renal Ischemia | 227 |
| Ch. 16 | Human Keratinocyte Migration Involves Extracellular Matrix and Soluble Factors | 251 |
| Ch. 17 | Endothelial Cell Signaling during Wound Healing | 259 |
| Ch. 18 | Development and Optimization of Preservation Solutions | 289 |
| Ch. 19 | Hypoxia, Free Radicals, and Reperfusion Injury Following Cold Storage and Reperfusion of Livers for Transplantation | 299 |
| Ch. 20 | Cellular Injury Associated with Organ Cryopreservation: Chemical Toxicity and Cooling Injury | 333 |
| Index | 357 |
| Price | Condition | Delivery | Seller | Action |
| $99.99 | Digital |
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S. Meston
reviewed Cell Biology of Trauma on September 15, 2014
Cell Biology of Trauma
I don't see that the eugenics movement moved much beyond this foundational document, either in politics or in scientific understanding, which is I suppose both a compliment to Galton and a judgment on his successors.
He should have said more about the nephews of Popes, which looked like the most promising piece of evidence in considering the basic nature/nurture issue.
I think implicitly it looks Galton wanted to turn humanity into a super-organism, like an ant-hill or bee hive. This might still be in the cards.
Also, I wanted to add that Galton displays what seems to me a sophisticated understanding of fitness landscapes and "evolution by jerks", even if only in a couple paragraphs near the end.
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